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Gender does not appear to be related to the degree of genetic influence on reading deficits (DeFries, Gillis, & Wadsworth, 1993), Nonsignificant trends in relation to age reported by Wadsworth, Gillis, DeFries, and Fulker (1989) have been further explored by DeFries et al. (1997). The latter study found trends toward decreasing heritability with age for word recognition and increasing heritability with age for spelling. The opposing direction of these trends resulted in a statistically significant interaction.

Visual perception: The neurophysiological foundations (pp. 163–203). San Diego: Academic Press. Livingstone, M. , Rosen, G. , Drislane, F. , & Galaburda, A. M. (1991). Physiological and anatomical evidence for a magnocellular defect in developmental dyslexia. Proceedings of the National Academy of Science, USA, 88, 7943–7947. , & Slaghuis, W. (1986). A theoretical and experimental case for a visual deficit in specific reading disability. Cognitive Neuropsychology, 3, 225–267. Meares, O. (1980).

2000). These authors argued that “a basic weakness in phonological representations ... limits dyslexic readers’ ability to build efficient structures within the angular gyrus that link orthographic codes computed in the extra-striate areas of the occipital lobe to phonological codes represented in the superior temporal gyrus” (p. 54–55). In a much quoted study, Paulesu et al. (1996) observed that, in comparison with controls, a small group of adult (compensated) dyslexic men with residual phonological processing difficulties showed reduced PET activation of the left insular region.

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